By Dr. Edward Yardeni , Amalia F. Quintana
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Additional resources for Asset Valuation & Allocation
The O allele produces no transferase, so the H antigen remains unmodiﬁed. The ABO gene on chromosome 9 consists of seven exons. The A1 and B alleles differ by seven nucleotides in exons 6 and 7, which encode a total of four amino acid substitutions at positions 176, 235, 266 and 268 of their glycosyltransferase products (Fig. 3). It is primarily the amino acids at positions 266 and 268 that determine whether the gene product is a GalNAc-transferase (A) or Gal-transferase (B). The most common O allele (O1) has an identical sequence to A1, apart from a single nucleotide deletion in exon 6, which shifts the reading frame and introduces a translation stop codon before the region of the catalytic site, so that any protein produced would be truncated and have no enzyme activity.
Cell-mediated immunity Antigen-speciﬁc cell-mediated responses are carried out by T cells. e. cytotoxicity). In addition they release cytokines that can trigger inﬂammation and are responsible for delayed hypersensitivity and symptoms usually associated with infection such as fever, myalgia and fatigue. Cytotoxic T cells Cytotoxicity is the job of cytotoxic T cells, which are distinguished by the presence of CD8 on the cell surface. This facilitates their interaction with HLA class I on the surface of cells altered by the presence of antigen, usually as the result of virus infection.
Cytotoxic T cells are the main means of protection against virus infection. They are also important mediators of allograft rejection. Like the B-cell response, the cytotoxic T-cell response requires help from T-helper cells and is regulated by these cells. Delayed hypersensitivity Delayed hypersensitivity is an example of another crucial role for helper T cells. It is dependent on the secretion of the cytokines interleukin (IL)-1, IL-2, tumour necrosis factor (TNF) and IFN-g, the socalled proinﬂammatory or Th1-type cytokines.
Asset Valuation & Allocation by Dr. Edward Yardeni , Amalia F. Quintana