By Michael H. Weisman MD, John D. Reveille, Desiree van der Heijde

ISBN-10: 0323070426

ISBN-13: 9780323070423

Constructed as a significant other textual content to the third variation of the heralded Rheumatology, by way of Hochberg et al., this cutting-edge reference provides state-of-the-art figuring out of the spondylarthropathies. you can find thorough examinations of the cutting edge, new remedies that problem older innovations of remedy and administration of the illness. famous specialists within the box additionally current the newest strategies in early prognosis utilizing complex imaging recommendations * state of the art wisdom of the pathogenetic and epidemiologic positive aspects * crucial new advancements in immunology * and the latest knowing of the socio-demographic effect of the disease.

  • Describes leading edge remedy and administration ideas that show you how to offer sufferers with monstrous aid in soreness, morbidity, and the extra dire outcomes of the affliction.
  • allows fast connection with the disease's actual manifestations via complete insurance of its medical features.
  • helps you to be sure the efficacy of organic brokers in line with the most recent insights on new treatments, together with anti-TNF.
  • Assesses the advantages and obstacles of accessible imaging modalities within the prognosis and administration of spondylarthropathies.

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Extra info for Ankylosing spondylitis and the spondyloarthropathies

Sample text

23 HLA-B27 and ankylosing spondylitis HLA-B27, which is encoded in the MHC class I region, confers the greatest known risk for AS and is found in more than 90% of patients of European ancestry with AS (reviewed in references. 24 and 25). 26 HLA-B27 is found in 60% to 70% of patients with psoriatic spondylitis and in 25% of those with peripheral psoriatic arthritis (PsA). Up to 70% of those with irritable bowel disease (IBD)-associated spondylitis have HLA-B27, although no HLA-B27 association is seen with asymptomatic sacroiliitis.

107. Brown MA, Brophy S, Bradbury L, et al. Identification of major loci controlling clinical manifestations of ankylosing spondylitis. Arthritis Rheum. 2003;48:2234–2239. 108. van der Linden SJ, van der Heijde D. Spondylarthopathies. Ankylosing spondylitis. In: Ruddy S, Harris ED, Sledge CB, eds. Kelley’s Textbook of Rheumatology. 6th ed. Philadelphia, Pa: Saunders;2000:1039–1053. 109. Nikkari S, Rantakokko K, Ekman P, et al. Salmonella-triggered reactive arthritis: use of polymerase chain reaction, immunocytochemical staining, and gas chromatography-mass spectrometry in the detection of bacterial components from synovial fluid.

Brocke P, Garbi N, Momburg F, Hammerling GJ. HLA-DM, HLADO and tapasin: functional similarities and differences. Curr Opin Immunol. 2002;14:22–29. 16. Deshaies F, Brunet A, Diallo DA, Denzin LK, Samaan A, Thibodeau J. A point mutation in the groove of HLA-DO allows egress from the endoplasmic reticulum independent of HLADM. Proc Natl Acad Sci USA. 2005;102:6443–6448. 17. Paulsson KM. Evolutionary and functional perspectives of the major histocompatibility complex class I antigen-processing machinery.

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Ankylosing spondylitis and the spondyloarthropathies by Michael H. Weisman MD, John D. Reveille, Desiree van der Heijde


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